Electronic Theses and Dissertations

Date of Award

1-1-2025

Document Type

Thesis

Degree Name

M.S. in Biological Science

First Advisor

Mika Jekabsons

Second Advisor

Bradley Jones

Third Advisor

Joshua Bloomekatz

School

University of Mississippi

Relational Format

dissertation/thesis

Abstract

This study investigates Bax binding to voltage-dependent anion-selective channels (VDAC1 and VDAC2) N-terminal 26-mer peptides, which have strong sequence homology to BH3 domains, to assess binding stoichiometries, affinities, residues involved in binding, and the potential role of peptide secondary structure in this process. Peptide helical content was assessed without or with Fos12 detergent using circular dichroism spectroscopy. Fluorescent-tagged peptides were used to assess binding affinities and stoichiometries to recombinant Bax, after correction for non-specific binding using bovine serum albumin. Affinities for VDAC1 and VDAC2 were three- to six-fold, respectively, lower than for the Bim BH3 domain, but all three peptides bound with a stoichiometry of one site per monomer. All three wild type peptides displayed ~67 % helical content with Fos12 present, whereas secondary structure was moderately to substantially reduced when mutations were introduced. Reduction in helical content strongly correlated with decreased affinity for a subset of peptides, whereas others exhibited affinities that were either strictly due to the introduced sequence modification, or a combination of the sequence modification and loss of secondary structure. Notably, the conserved aspartate (D16 or D27 for human VDACs 1 & 2, respectively) was important for Bax binding. Some double hydrophobic residue VDAC mutants bound with a stoichiometry of two sites per monomer, supporting previous work indicating two distinct BH3 domain sites on Bax. The results are consistent with the possibility that VDACs 1 & 2 interact with Bax through their N-terminal BH3-like domains.

Available for download on Friday, January 30, 2026

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